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Background and Objective The most recently approved biologics for moderate-to-severe psoriasis are the interleukin (IL)-17 and IL-23 inhibitors. Drug survival is a frequently used outcome to assess drug performance in practice. An overview of the available drug survival studies regarding IL-17 and IL-23 inhibitors is lacking. Therefore, our objective was to assess the drug survival of IL-17
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Given the strong pro-inflammatory role of IL-17, agents that target IL-17 or the IL-17 receptor (IL-17R) are attractive candidates for inflammatory diseases. a small molecule drug that
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IL-17 inhibitors are research hotspots, for IL-17 (interleukin 17) plays a role in numerous immune-mediated disorders, such as rheumatoid arthritis, Crohn's
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Both IL-23 and IL-17 inhibitors are effective in treating psoriasis. However, IL-23 is a more advanced concept than IL-17. IL-23 is an important
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Therapies such as secukinumab, ixekizumab, and brodalumab inhibit IL-17 subtypes. For example, brodalumab inhibits the downstream effects of all five IL-17 subtypes (IL-17A, IL-17AF, IL-17F, IL-17C, and IL-17E) via IL-17 receptor blockade; brodalumab can also normalize the levels of IL-23.
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The proinflammatory properties of IL-17 are protective against infection. A good understanding of the different roles played by the IL-17 cytokines is necessary to design effective drugs relating to pathogenesis and mechanisms of inflammation. Unrestrained IL-17 signalling is associated with immunopathology, autoimmune disease, and cancer
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Lists IL-17 and IL-17 Receptor Inhibitor Drugs along with their targets, conditions approved for, and/or clinical trial status. Role of IL-17 in autoimmunity The IL-17 family represents a distinct and complex cytokine signaling system that is highly conserved across vertebrate evolution [ 1 3 ].
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In conclusion, we have discussed the IL-17 family and their receptors in the development of pancreatitis, T1DM, and PC. The application of drug therapy targeting IL-17 in pancreatic diseases is also summarized. However, the current reports on IL-17 in pancreatic diseases mainly focus on IL-17A, with a few involving IL-17B and IL-17E.
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Abstract. Interleukin 17 (IL-17) is described as a pro-inflammatory cytokine produced by activated CD4 cells. Evidence shows that IL-17 is highly up-regulated at sites of inflammatory tissues of autoimmune diseases and amplifies the inflammation through synergy with other cytokines, such as TNF (tumor necrosis factor) . Therefore, targeting IL-17/IL-17R, IL-17-producing pathways or IL-17
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